Gout

Purines (adenine, guanine)

  • are metabolised to hypoxanthine
  • changed by xanthine oxidase to xanthine
  • Further metabolised to uric acid

Pyrimidines (thymine, cytosine)

  • are metabolised to ammonium salts + urea.

Nucleoside = Base + ribose: Eg. RNA
Nucleotide = Base + ribose + phosphate radical

 

Classification of hyperuricaemia

  1. Primary: absolute/relative abnormality of xanthine-hypoxanthine handling
    • Deficiency of PPRT (Lesch-Nyhan syndrome) leads to inability of xanthine/hypoxanthine to be recycled into purines
  2. Secondary: increased purine breakdown with increased formation of uric acid
    • Ingestion - caviar, roe
    • Increased cell turnover: psoriasis, sickle cell, leukaemia, malignancy
    • Decreased excretion: CRF, diuretics

Complications of gout

  • Joints: destructive osteoarthropathy
  • Renal tract  - stones, renal failure from a big stone